User:Mr. Ibrahem/Posterior urethral valve1
Medical condition / From Wikipedia, the free encyclopedia
Posterior urethral valve (PUV) is a disorder were the urethra in a newborn male is blocked by a fold of tissue.[1] Symptoms after birth may include poor feeding or failure to thrive.[1] It may be associated with urinary retention, chronic kidney disease, incomplete development of the lungs, urinary incontinence, or urinary tract infections.[1]
Posterior urethral valve | |
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Other names | Congenital obstructive posterior urethral membrane |
Vesiculæ seminales and ampullæ of ductus deferentes, seen from the front. Posterior urethral valves are at the dorsal aspect (back) of the prostatic urethra. | |
Specialty | Urology |
Symptoms | Poor feeding, failure to thrive[1] |
Complications | Chronic kidney disease, incomplete lung development, urinary incontinence, urinary tract infections[1] |
Usual onset | Present at birth[1] |
Types | Type I, II, III[1] |
Causes | Unknown[2] |
Risk factors | Family history.[2] |
Diagnostic method | Ultrasound, voiding cystourethrography[1][2] |
Differential diagnosis | Other causes of oligohydramnios, vesicoureteral reflux, Prune–belly syndrome, ureteric stenosis[1] |
Treatment | Urinary catheter, surgery, management of complications[1] |
Prognosis | Variable[1] |
Frequency | 1 in 5,000 male births[1] |
How and why the disorder forms in unclear.[2] Risk factors include a family history.[2] They can be divided into three types based on the proposed origin of the fold of tissue.[1] The potential origins of the fold; however, is disputed as of 2020.[1] Diagnosis may occur before birth by ultrasound or after birth via voiding cystourethrography.[1]
Surgery may be carried out before birth; however, results are mixed.[1] After birth the bladder may be drained with a feeding tube.[1] Other efforts may be required to address electrolyte or breathing problems.[1] Surgery is required to remove the fold of tissue and potentially to treat any vesicoureteric reflux.[1]
Posterior urethral valve occurs in about one in 5,000 male births.[1] The condition was first described in 1717 by Giovanni Battista Morgagni.[1] The classification into types occurred in 1919 by Hugh Young.[3]